Box 951740, 27-117 CHS
Los Angeles, CA 90095-1740
On-campus mail: 174017
Tel: (310) 206-6575
M.D., Ph.D., Professor and Executive Chair, Departments of
Obstetrics and Gynecology, and Professor, Department of Molecular
and Medical Pharmacology Westwood Campus, Program Director, UCLA
Dr. Chaudhuri completed his MBBS from Calcutta University, India
and his M.D. from the All-India Institute of Medical Sciences, India.
He then obtained a Ph.D. in Pharmacology from London University
under the mentorship of Professor Sir John Vane, who won the Nobel
Prize in Medicine or Physiology in 1982. He subsequently completed
his residency in Obstetrics and Gynecology from the State University
of New York at Buffalo and joined UCLA to do a fellowship in Reproductive
Endocrinology and Infertility. Following completion of this, he
joined the department as an Assistant Professor. Since joining UCLA,
he has published extensively on the action of sex steroids and nitric
oxide. He, together with Lou Ignarro, Professor of Pharmacology
at UCLA and a Nobel Laureate, was a member of one of the first groups
to demonstrate that endothelium-derived relaxing factor is nitric
oxide. He has in the past and continues at present to be supported
by the NIH in the form of two RO1 grants. He has served for 4 years
as a member of the Human Embryology and Development Review Group
of the NIH program project review groups and for 4 years as a member
of the Maternal and Child Health Committee of the NICHD. Recently,
he was appointed as a member of the Advisory Council of the NICHD.
Dinh H, Chaudhuri G, Nathan L. Testosterone attenuates expression
of Vascular Cell Adhesion Molecule-1 by conversion to Estradiol
by Aromatase in Endothelial cells: Implications in Atherosclerosis.
Proc Natl Acad Sci USA 2002;99:4055-4060.
Singh R, Pervin
S, Chaudhuri G. Caspase -8 mediated BID cleavage and release
of mitochondrial cytochrome c during N?- hydroxy L-arginine induced
apoptosis in MDA-MB-468 cells: antagonistic effects of L-ornithine.
J Biol Chem 2002;227:37630-37636.
Nathan L, Dinh H, Reddy ST, Chaudhuri G. 17-Epiestriol, an
estrogen metabolite, is more potent than Estradiol in inhibiting
VCAM-1 mRNA expression. J Biol Chem 2003;278:11746-11752.